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The mechanism is largely angiogenic: BPC-157 upregulates VEGF (vascular endothelial growth factor) and activates VEGFR2, stimulating the formation of new blood vessels in damaged tissue

This does not however apply for ER-positive breast cancer cell line MCF7, T47D and ZR-75, which may be explained by the intrinsic characteristics of the molecular statuses of different breast cancer cell lines [60, 65]

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